The heat conduction of the necrotic skin decreases after burn. Moreover, heat exchange between the skin and the outer air can’t be achieved because the sweat gland is damaged.

However, the heat conductivity of the tissues beneath the necrotic tissues remains nearly at the normal level so the remnant heat will conduct to the inner layer to cause secondary heat injury.

The peripheral circulation doesn’t relieve effectively after burn and the local metabolic products can’t be discharged in time, which will cause the progressive ischemia and hypoxia that will lead to necrosis.

And more over, the normal skin structures are damaged so that the moisture evaporation capacity on the burn increases sharply, which will promote the permeability of the capillary and the exosmose the blood plasma. In the end, the microcirculation obstacle will be aggravated.

The decomposed and rancid necrotic skin is the good culture for bacterium so the amount of bacterium increases exponentially. The bacterial content in the tissues beneath the scab will reach
to 105/g in 48 hours without intervention.

And the wound sepsis will invade the stasis zone and the normal tissues to produce large amount of toxins, which will lead to systemic toxaemia.

For traditional dry exposed therapy, cold treatment using cold water is adopted after burn.

The burn wound baking to promote the crust formation will not only cause the secondary heat injury to the burn wound, but also the frequent contraction and expansion of the capillary aggravates the microcirculation obstacle that leads to the irreversible necrosis of the stasis zone.

The complete debridement to the burn wound, the damage of the detergent to the wound, the rough operation、the non-in time dressing changing and hemorrhage are the iatrogenic reasons that will cause secondary damage to the burn wound. We must pay attention to it.

The melting point of MEBO is low and the heat conductivity of it is stronger than normal skin tissue, so the direction of the heat flow changes.

The remnant heat of the burn wound conducts to
MEBO but not to the normal tissues. Heat absorbed MEBO melts, so that the proportion decreases.

The melted MEBO floats on the burn wound to bring away the remnant heat to prevent the secondary heat damage.

MEBO can keep the wound in a physiologically moist environment (See Q14, Part1), which will maximally reduce the amount of moisture evaporation on the burn wound to relieve the pachemia of the stasis zone. Further damage to the burn wound is prevented.

The research shows that the shapes of kinds of bacterium existed on the burn wound will vary under the action of MEBO, which will lower the productivity and the invasive power.

It’s very effective for local infection control at the early stage after burn.

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